Researcher Portfolio
Moritz, Ralf
Dept. Müllen: Synthetic Chemistry, MPI for Polymer Research, Max Planck Society
Researcher Profile
Position: Dept. Müllen: Synthetic Chemistry, MPI for Polymer Research, Max Planck Society
Researcher ID: https://pure.mpg.de/cone/persons/resource/persons128509
Publications
: Hallumi, E., Shalah, R., Lo, W. L., Corso, J., Oz, I., Beach, D., Wittman, S., Isenberg, A., Sela, M., Urlaub, H., Weiss, A., & Yablonski, D. (2021). Itk promotes the integration of TCR and CD28 costimulation through its direct substrates SLP-76 and Gads. The Journal of Immunology, 206(10), 2322-2337. doi:10.4049/jimmunol.2001053. [PubMan] : Mohr, S., Doebele, C., Comoglio, F., Berg, T., Beck, J., Bohnenberger, H., Alexe, G., Corso, J., Ströbel, P., Wachter, A., Beissbarth, T., Schnutgen, F., Cremer, A., Haetscher, N., Göllner, S., Rouhi, A., Palmqvist, L., Rieger, M. A., Schroeder, T., Bönig, H., Müller-Tidow, C., Kuchenbauer, F., Schütz, E., Green, A. R., Urlaub, H., Stegmaier, K., Humphries, R. K., Serve, H., & Oellerich, T. (2017). Hoxa9 and Meis1 cooperatively induce addiction to Syk signaling by suppressing miR-146a in acute myeloid leukemia. Cancer Cell, 31(4), 549-562. doi:10.1016/j.ccell.2017.03.001. [PubMan] : Manno, B., Oellerich, T., Schnyder, T., Corso, J., Lösing, M., Neumann, K., Urlaub, H., Batista, F. D., Engelke, M., & Wienands, J. (2016). The Dok-3/Grb2 adaptor module promotes inducible association of the lipid phosphatase SHIP with the BCR in a coreceptor-independent manner. European Journal of Immunology, 46(11), 2520-2530. doi:10.1002/eji.201646431. [PubMan] : Walter, R., Corso, J., Pan, K. T., Doebele, C., Yepes, D., Sellner, L., Tomska, K., Bohnenberger, H., Zenz, T., Urlaub, H., Serve, H., & Oellerich, T. (2016). Elucidation of tonic and activated B cell receptor signaling in Burkitt's lymphoma reveals insights into non-oncogene addiction. Oncology Research and Treatment, 39(Suppl.), 219-219. [PubMan] : Corso, J., Pan, K. T., Walter, R., Doebele, C., Mohr, S., Bohnenberger, H., Ströbel, P., Lenz, C., Slabicki, M., Hüllein, J., Comoglio, F., Rieger, M. A., Zenz, T., Wienands, J., Engelke, M., Serve, H., Urlaub, H., & Oellerich, T. (2016). Elucidation of tonic and activated B-cell receptor signaling in Burkitt's lymphoma provides insights into regulation of cell survival. Proceedings of the National Academy of Sciences of the United States of America, 113(20), 5688-5693. doi:10.1073/pnas.1601053113. [PubMan] : Corso, J. (2016). Quantitative phosphoproteomics for studying B-cell receptor signaling in Burkitt's lymphoma. PhD Thesis, Georg-August-Universität, Göttingen. [PubMan] : Doebele, C., Corso, J., Cremer, A., Muench, S., Beck, J., Lenz, C., Bohnenberger, H., Wachter, A., Beissbarth, T., Schutz, E., Serve, H., Urlaub, H., & Oellerich, T. (2015). Elucidation of B cell receptor signaling in Burkitt's lymphoma reveals novel signaling nodes with potential therapeutic relevance. Cancer Research, 75(Suppl. 15): 50. [PubMan] : Kuvardina, O. N., Herglotz, J., Kolodziej, S., Kohrs, N., Herkt, S., Wojcik, B., Oellerich, T., Corso, J., Behrens, K., Kumar, A., Hussong, H., Urlaub, H., Koch, J., Serve, H., Bonig, H., Stocking, C., Rieger, M. A., & Lausen, J. (2015). RUNX1 represses the erythroid gene expression program during megakaryocytic differentiation. Blood, 125(23), 3570-3579. doi:10.1182/blood-2014-11-610519. [PubMan] : Oellerich, T., Corso, J., Beck, J., Pan, K. T., Doebele, C., Wachter, A., Lenz, C., Beissbarth, T., Schuetz, E., Tomska, K., Sellner, L., Zenz, T., Serve, H., & Urlaub, H. (2015). The B cell reseptor signaling output in Burkitt´s lymphoma is genotype specific and impacts sensitivity towards BCR signaling inhibitors. Haematologica, 100(Suppl. 1), 178-178. [PubMan] : Oellerich, T., Corso, J., Beck, J., Doebele, C., Wachter, A., Lenz, C., Bohnenberger, H., Beissbarth, T., Gökbuget, N., Schutzatz, E., Serve, H., & Urlaub, H. (2015). Elucidation of B cell receptor signaling in Burkitt's lymphoma reveals novel signaling nodes with potential therapeutic relevance. Clinical Lymphoma Myeloma and Leukemia, 15(Suppl. 1), S226-S227. [PubMan] : Oellerich, T., Mohr, S., Corso, J., Beck, J., Döbele, C., Braun, H., Cremer, A., Münch, S., Wicht, J., Oellerich, M. F., Bug, G., Bohnenberger, H., Perske, C., Schütz, E., Urlaub, H., & Serve, H. (2015). FLT3-ITD and TLR9 use Bruton tyrosine kinase to activate distinct transcriptional programs mediating AML cell survival and proliferation. Blood, 125(12), 1936-1947. doi:10.1182/blood-2014-06-585216. [PubMan] : Bohnenberger, H., Ströbel, P., Mohr, S., Corso, J., Berg, T., Urlaub, H., Lenz, C., Serve, H., & Oellerich, T. (2015). Quantitative mass spectrometric profiling of cancer-cell proteomes derived from liquid and solid tumors. Journal of Visualized Experiments, 96: e52435. doi:10.3791/52435. [PubMan] : Lugassy, J., Corso, J., Beach, D., Petrik, T., Oellerich, T., Urlaub, H., & Yablonski, D. (2015). Modulation of TCR responsiveness by the Grb2-family adaptor, Gads. Cellular Signalling, 27(1), 125-134. doi:10.1016/j.cellsig.2014.10.005. [PubMan] : Oellerich, T., Mohr, S., Beck, J., Corso, J., Bohnenberger, H., Döbele, C., Burg, G., Schütz, E., Urlaub, H., & Serve, H. (2014). Syk and Btk induce cell survival and proliferation in AML cells by activating distinct signaling pathways and transcriptional programs. Oncology Research and Treatment, 37(Suppl. 5), 225-226. [PubMan] : Bohnenberger, H., Ströbel, P., Henric-Petri, H., Lenz, C., Emmert, A., Bremmer, F., Strecker, J., Holland, R., Hinterthaner, M., Corso, J., Wagner, S., Küffer, S., Sebastian, M., Bergmann, L., Danner, B., Schöndube, F. A., Serve, H., Urlaub, H., & Oellerich, T. (2014). Comprehensive quantitative proteomic profiling of lung cancers reveals novel biomarkers and potential drug targets. Cancer Research, 74(Supplement S): 2487. [PubMan] : Oellerich, T., Mohr, S., Beck, J., Bohnenberger, H., Doebele, C., Corso, J., Burg, G., Schütz, E., Urlaub, H., & Serve, H. (2014). Syk and Btk transduce survival and proliferation-inducing signals by activating distinct signaling pathways and transcriptional programs in AML cells. Blood, 124(21). [PubMan] : Oellerich, T., Oellerich, M. F., Engelke, M., Münch, S., Mohr, S., Nimz, M., Hsiao, H. H., Corso, J., Zhang, J., Bohnenberger, H., Berg, T., Rieger, M. A., Wienands, J., Bug, G., Brandts, C., Urlaub, H., & Serve, H. (2013). β2 integrin–derived signals induce cell survival and proliferation of AML blasts by activating a Syk/STAT signaling axis. Blood, 121(19), 3889-3899. doi:10.1182/blood-2012-09-457887. [PubMan] : Urlaub, H., Corso, J., Oellerich, T., & Serve, H. (2013). Quantitative mass spectrometry: Elucidation of the transduction cascade triggered by B cell receptors. GIT Laboratory Journal Europe, 17(1-2), 22-24. [PubMan] : Oellerich, T., Oellerich, M. F., Engelke, M., Münch, S., Mohr, S., Nimz, M., Hsiao, H. H., Corso, J., Zhang, J., Bohnenberger, H., Berg, T., Rieger, M. A., Wienands, J., Bug, G., Brandts, C., Urlaub, H., & Serve, H. (2013). β2 integrin–derived signals induce cell survival and proliferation of AML blasts by activating a Syk/STAT signaling axis. Cancer Research, 73(Supplement 1): 1107. [PubMan]