Researcher Portfolio
Stehle, Thomas
Metchnikoff Laboratory, Max Planck Institute of Immunobiology and Epigenetics, Max Planck Society
Researcher Profile
Position: Metchnikoff Laboratory, Max Planck Institute of Immunobiology and Epigenetics, Max Planck Society
Researcher ID: https://pure.mpg.de/cone/persons/resource/persons191330
Publications
: Sheikh, B. N., Guhathakurta, S., Tsang, T. H., Schwabenland, M., Renschler, G. V., Herquel, B., Bhardwaj, V., Holz, H., Stehle, T., Bondareva, O., Aizarni, N., Mossad, O., Kretz, O., Reichardt, W., Chatterjee, A., Braun, L. J., Thevenon, J., Sartelet, H., Blank, T., Grün, D., von Elverfeldt, D., Huber, T. B., Vestweber, D., Avilov, S., Prinz, M., Büscher, J. M., & Akhtar, A. (2020). Neural metabolic imbalance induced by MOF dysfunction triggers pericyte activation and breakdown of vasculature. Nature Cell Biology, 22, 828-841. doi:org/10.1038/s41556-020-0526-8. [PubMan] : Rodrigues, C. P., Herman, J. S., Herquel, B., Velsecchi Keller, C. I., Stehle, T., Grün, D., & Akhtar, A. (2020). Temporal expression of MOF acetyltransferase primes transcription factor networks for erythroid fate. Science Advances, 6, eaaz4815. doi:10.1126/sciadv.aaz4815. [PubMan] : Sheikh, B., Bechtel-Walz, W., Lucci, J., Karpuik, O., Hild, I., Hartleben, B., Vornweg, J., Helmstädter, M., Sahyoun, A., Bhardwaj, V., Stehle, T., Diehl, S., Kretz, O., Voss, A., Thomas, T., Manke, T., Huber, T., & Akhtar, A. (2016). MOF maintains transcriptional programs regulating cellular stress response. Oncogene, 35, 2698-2710. doi:doi: 10.1038/onc.2015.335. [PubMan] : Nassal, M., Skamel, C., Vogel, M., Kratz, P. A., Stehle, T., Wallich, R., & Simon, M. M. (2007). Development of hepatitis B virus capsids into a whole-chain protein antigen display platform: New particulate Lyme disease vaccines. International Journal of Medical Microbiology, 298, 135-142. [PubMan] : Skamel, C., Ploss, M., Böttcher, B., Stehle, T., Wallich, R., Simon, M. M., & Nassal, M. (2006). Hepatitis B Virus Capsid-like Particles Can Display the Complete, Dimeric Outer Surface Protein C and Stimulate Production of Protective Antibody Responses against Borrelia burgdorferi Infection. The Journal of Biological Chemistry, 281, 17474-17481. [PubMan] : Glenz, K., Bouchon, B., Stehle, T., Wallich, R., Simon, M. M., & Warzecha, H. (2006). Production of a recombinant bacterial lipoprotein in higher plant chloroplasts. Nature Biotechnology, 24, 76-77. [PubMan] : Nassal, M., Skamel, C., Kratz, P. A., Wallich, R., Stehle, T., & Simon, M. M. (2005). A fusion product of the complete Borrelia burgdorferi outer surface protein A (OspA) and the hepatitis B virus capsid protein is highly immunogenic and induces protective immunity similar to that seen with an effective lipidated OspA vaccine formula. European Journal of Immunology, 35, 655-665. [PubMan] : Lederer, S., Brenner, C., Stehle, T., Gern, L., Wallich, R., & Simon, M. M. (2004). Quantitative analysis of Borrelia burgdorferi gene expression in naturally (tick) infected mouse strains. Medical Microbiology and Immunology, 194, 81-90. [PubMan] : Rode, M., Balkow, S., Sobek, V., Brehm, R., Martin, P., Kersten, A., Dumrese, T., Stehle, T., Müllbacher, A., Wallich, R., & Simon, M. M. (2004). Perforin and Fas Act Together in the Induction of Apoptosis, and Both Are Critical in the Clearance of Lymphocytic Choriomeningitis Virus Infection. Journal of Virology, 78, 12395-12405. [PubMan] : Wallich, R., Jahraus, O., Stehle, T., Tran, T. T. T., Brenner, C., Hofmann, H., Gern, L., & Simon, M. M. (2003). Artificial-infection protocols allow immunodetection of novel Borrelia burgdorferi antigens suitable as vaccine candidates against Lyme disease. European Journal of Immunology, 33(3), 708-719. [PubMan] : Müllbacher, A., Lobigs, M., Hla, R. T., Tran, T., Stehle, T., & Simon, M. M. (2002). Antigen-Dependent Release of IFN-γ by Cytotoxic T Cells Up- Regulates Fas on Target Cells and Facilitates Exocytosis- Independent Specific Target Cell Lysis. Journal of Immunology, 169(1), 145-150. [PubMan]