Researcher Portfolio

 
   

Li, Xiang

Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society  

 

Researcher Profile

 
Position: Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society
Researcher ID: https://pure.mpg.de/cone/persons/resource/persons228662

External references

 

Publications

 
 
 : Wu, F., Li, X., Looso, M., Liu, H., Ding, D., Guenther, S., Kuenne, C., Liu, S., Weissmann, N., Boettger, T., Atzberger, A., Kolahian, S., Renz, H., Offermanns, S., Gaertner, U., Potente, M., Zhou, Y., Yuan, X., & Braun, T. (2024). Spurious transcription causing innate immune responses is prevented by 5-hydroxymethylcytosine. NATURE GENETICS, 55(1), 100-+. doi:10.1038/s41588-022-01252-3. [PubMan] : Li, X., Wu, F., Guenther, S., Looso, M., Kuenne, C., Zhang, T., Wiesnet, M., Klatt, S., Zukunft, S., Fleming, I., Poschet, G., Wietelmann, A., Atzberger, A., Potente, M., Yuan, X., & Braun, T. (2023). Inhibition of fatty acid oxidation enables heart regeneration in adult mice. NATURE. doi:10.1038/s41586-023-06585-5. [PubMan] : Wu, F., Li, X., Looso, M., Liu, H., Ding, D., Guenther, S., Kuenne, C., Liu, S., Weissmann, N., Boettger, T., Atzberger, A., Kolahian, S., Renz, H., Offermanns, S., Gaertner, U., Potente, M., Zhou, Y., Yuan, X., & Braun, T. (2023). Spurious transcription causing innate immune responses is prevented by 5-hydroxymethylcytosine. NATURE GENETICS, (2023; 55(1)), 100-111. doi:10.1038/s41588-022-01252-3. [PubMan] : Garcia-Gonzalez, C., Dieterich, C., Maroli, G., Wiesnet, M., Wietelmann, A., Li, X., Yuan, X., Graumann, J., Stellos, K., Kubin, T., Schneider, A., & Braun, T. (2022). ADAR1 Prevents Autoinflammatory Processes in the Heart Mediated by IRF7. CIRCULATION RESEARCH, 131(7), 580-597. doi:10.1161/CIRCRESAHA.122.320839. [PubMan] : Yuan, X., Qi, H., Li, X., Wu, F., Fang, J., Bober, E., Dobreva, G., Zhou, Y., & Braun, T. (2017). Disruption of spatiotemporal hypoxic signaling causes congenital heart disease in mice. JOURNAL OF CLINICAL INVESTIGATION, 127(6), 2235-2248. doi:10.1172/JCI88725. [PubMan]