Researcher Portfolio

 
   

Feigenbutz, Dennis

IMPRS-BI: Martinsried, Martinsried, Max Planck Institute for Biological Intelligence, Max Planck Society, Research Group: Molecular Neurodegeneration / Dudanova, Martinsried, Max Planck Institute for Biological Intelligence, Max Planck Society, Research Group: Molecular Neurodegeneration / Dudanova, MPI of Neurobiology, Max Planck Society  

 

Researcher Profile

 
Position: Research Group: Molecular Neurodegeneration / Dudanova, MPI of Neurobiology, Max Planck Society
Position: Research Group: Molecular Neurodegeneration / Dudanova, Martinsried, Max Planck Institute for Biological Intelligence, Max Planck Society
Position: IMPRS-BI: Martinsried, Martinsried, Max Planck Institute for Biological Intelligence, Max Planck Society
Researcher ID: https://pure.mpg.de/cone/persons/resource/persons268288

External references

 
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Publications

 
 
 : Sitron, C. S., Trinkaus, V. A., Galesic, A., Garhammer, M., Yuste-Checa, P., Dransfeld, U., Feigenbutz, D., Zhang, J., Dudanova, I., Harper, J. W., & Hartl, F. U. (2025). α-Synuclein aggregates inhibit ESCRT-III through sequestration and collateral degradation. bioRxiv. doi:10.1101/2025.01.13.632710. [PubMan] : Sitron, C. S., Trinkaus, V. A., Galesic, A., Garhammer, M., Checa, P. Y., Dransfeld, U., Feigenbutz, D., Zhang, J., Dudanova, I., Harper, J. W., & Hartl, F. U. (2025). α-Synuclein aggregates inhibit ESCRT-III through sequestration and collateral degradation. bioRxiv: the preprint server for biology. doi:10.1101/2025.01.13.632710. [PubMan] : Voelkl, K., Gutiérrez-Ángel, S., Keeling, S., Koyuncu, S., da Silva Padilha, M., Feigenbutz, D., Arzberger, T., Vilchez, D., Klein, R., & Dudanova, I. (2023). Neuroprotective effects of hepatoma-derived growth factor in models of Huntington's disease. Life science alliance, 6(11): e202302018. doi:10.26508/lsa.202302018. [PubMan] : Riera Tur, I., Schäfer, T., Hornburg, D., Mishra, A., da Silva Padilha, M., Fernández-Mosquera, L., Feigenbutz, D., Auer, P., Mann, M., Baumeister, W., Klein, R., Meissner, F., Raimundo, N., Fernandez-Busnadiego, R., & Dudanova, I. (2022). Amyloid-like aggregating proteins cause lysosomal defects in neurons via gain-of-function toxicity. Life science alliance, 5(3): e202101185. doi:10.26508/lsa.202101185. [PubMan]