Researcher Portfolio

 
   

Tautenhahn, Susanne

Biod.AI.versity Observation & Integration (Bio.AI), Dr. Jana Waeldchen, Department Biogeochemical Integration, Prof. Dr. M. Reichstein, Max Planck Institute for Biogeochemistry, Max Planck Society, Functional Biogeography, Max Planck Institute for Biogeochemistry, Max Planck Society, Interdepartmental Max Planck Fellow Group Functional Biogeography, Max Planck Institute for Biogeochemistry, Max Planck Society, Research Group Organismic Biogeochemistry, Dr. C. Wirth, Max Planck Institute for Biogeochemistry, Max Planck Society  

 

Researcher Profile

 
Position: Interdepartmental Max Planck Fellow Group Functional Biogeography, Max Planck Institute for Biogeochemistry, Max Planck Society
Position: Functional Biogeography, Max Planck Institute for Biogeochemistry, Max Planck Society
Position: Biod.AI.versity Observation & Integration (Bio.AI), Dr. Jana Waeldchen, Department Biogeochemical Integration, Prof. Dr. M. Reichstein, Max Planck Institute for Biogeochemistry, Max Planck Society
Position: Research Group Organismic Biogeochemistry, Dr. C. Wirth, Max Planck Institute for Biogeochemistry, Max Planck Society
Researcher ID: https://pure.mpg.de/cone/persons/resource/persons62574

External references

 
WorldCat Search for Tautenhahn, Susanne
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Publications

 
 
 : Hallumi, E., Shalah, R., Lo, W. L., Corso, J., Oz, I., Beach, D., Wittman, S., Isenberg, A., Sela, M., Urlaub, H., Weiss, A., & Yablonski, D. (2021). Itk promotes the integration of TCR and CD28 costimulation through its direct substrates SLP-76 and Gads. The Journal of Immunology, 206(10), 2322-2337. doi:10.4049/jimmunol.2001053. [PubMan] : Mohr, S., Doebele, C., Comoglio, F., Berg, T., Beck, J., Bohnenberger, H., Alexe, G., Corso, J., Ströbel, P., Wachter, A., Beissbarth, T., Schnutgen, F., Cremer, A., Haetscher, N., Göllner, S., Rouhi, A., Palmqvist, L., Rieger, M. A., Schroeder, T., Bönig, H., Müller-Tidow, C., Kuchenbauer, F., Schütz, E., Green, A. R., Urlaub, H., Stegmaier, K., Humphries, R. K., Serve, H., & Oellerich, T. (2017). Hoxa9 and Meis1 cooperatively induce addiction to Syk signaling by suppressing miR-146a in acute myeloid leukemia. Cancer Cell, 31(4), 549-562. doi:10.1016/j.ccell.2017.03.001. [PubMan] : Manno, B., Oellerich, T., Schnyder, T., Corso, J., Lösing, M., Neumann, K., Urlaub, H., Batista, F. D., Engelke, M., & Wienands, J. (2016). The Dok-3/Grb2 adaptor module promotes inducible association of the lipid phosphatase SHIP with the BCR in a coreceptor-independent manner. European Journal of Immunology, 46(11), 2520-2530. doi:10.1002/eji.201646431. [PubMan] : Walter, R., Corso, J., Pan, K. T., Doebele, C., Yepes, D., Sellner, L., Tomska, K., Bohnenberger, H., Zenz, T., Urlaub, H., Serve, H., & Oellerich, T. (2016). Elucidation of tonic and activated B cell receptor signaling in Burkitt's lymphoma reveals insights into non-oncogene addiction. Oncology Research and Treatment, 39(Suppl.), 219-219. [PubMan] : Corso, J., Pan, K. T., Walter, R., Doebele, C., Mohr, S., Bohnenberger, H., Ströbel, P., Lenz, C., Slabicki, M., Hüllein, J., Comoglio, F., Rieger, M. A., Zenz, T., Wienands, J., Engelke, M., Serve, H., Urlaub, H., & Oellerich, T. (2016). Elucidation of tonic and activated B-cell receptor signaling in Burkitt's lymphoma provides insights into regulation of cell survival. Proceedings of the National Academy of Sciences of the United States of America, 113(20), 5688-5693. doi:10.1073/pnas.1601053113. [PubMan] : Corso, J. (2016). Quantitative phosphoproteomics for studying B-cell receptor signaling in Burkitt's lymphoma. PhD Thesis, Georg-August-Universität, Göttingen. [PubMan] : Doebele, C., Corso, J., Cremer, A., Muench, S., Beck, J., Lenz, C., Bohnenberger, H., Wachter, A., Beissbarth, T., Schutz, E., Serve, H., Urlaub, H., & Oellerich, T. (2015). Elucidation of B cell receptor signaling in Burkitt's lymphoma reveals novel signaling nodes with potential therapeutic relevance. Cancer Research, 75(Suppl. 15): 50. [PubMan] : Kuvardina, O. N., Herglotz, J., Kolodziej, S., Kohrs, N., Herkt, S., Wojcik, B., Oellerich, T., Corso, J., Behrens, K., Kumar, A., Hussong, H., Urlaub, H., Koch, J., Serve, H., Bonig, H., Stocking, C., Rieger, M. A., & Lausen, J. (2015). RUNX1 represses the erythroid gene expression program during megakaryocytic differentiation. Blood, 125(23), 3570-3579. doi:10.1182/blood-2014-11-610519. [PubMan] : Oellerich, T., Corso, J., Beck, J., Pan, K. T., Doebele, C., Wachter, A., Lenz, C., Beissbarth, T., Schuetz, E., Tomska, K., Sellner, L., Zenz, T., Serve, H., & Urlaub, H. (2015). The B cell reseptor signaling output in Burkitt´s lymphoma is genotype specific and impacts sensitivity towards BCR signaling inhibitors. Haematologica, 100(Suppl. 1), 178-178. [PubMan] : Oellerich, T., Corso, J., Beck, J., Doebele, C., Wachter, A., Lenz, C., Bohnenberger, H., Beissbarth, T., Gökbuget, N., Schutzatz, E., Serve, H., & Urlaub, H. (2015). Elucidation of B cell receptor signaling in Burkitt's lymphoma reveals novel signaling nodes with potential therapeutic relevance. Clinical Lymphoma Myeloma and Leukemia, 15(Suppl. 1), S226-S227. [PubMan] : Oellerich, T., Mohr, S., Corso, J., Beck, J., Döbele, C., Braun, H., Cremer, A., Münch, S., Wicht, J., Oellerich, M. F., Bug, G., Bohnenberger, H., Perske, C., Schütz, E., Urlaub, H., & Serve, H. (2015). FLT3-ITD and TLR9 use Bruton tyrosine kinase to activate distinct transcriptional programs mediating AML cell survival and proliferation. Blood, 125(12), 1936-1947. doi:10.1182/blood-2014-06-585216. [PubMan] : Bohnenberger, H., Ströbel, P., Mohr, S., Corso, J., Berg, T., Urlaub, H., Lenz, C., Serve, H., & Oellerich, T. (2015). Quantitative mass spectrometric profiling of cancer-cell proteomes derived from liquid and solid tumors. Journal of Visualized Experiments, 96: e52435. doi:10.3791/52435. [PubMan] : Lugassy, J., Corso, J., Beach, D., Petrik, T., Oellerich, T., Urlaub, H., & Yablonski, D. (2015). Modulation of TCR responsiveness by the Grb2-family adaptor, Gads. Cellular Signalling, 27(1), 125-134. doi:10.1016/j.cellsig.2014.10.005. [PubMan] : Oellerich, T., Mohr, S., Beck, J., Corso, J., Bohnenberger, H., Döbele, C., Burg, G., Schütz, E., Urlaub, H., & Serve, H. (2014). Syk and Btk induce cell survival and proliferation in AML cells by activating distinct signaling pathways and transcriptional programs. Oncology Research and Treatment, 37(Suppl. 5), 225-226. [PubMan] : Bohnenberger, H., Ströbel, P., Henric-Petri, H., Lenz, C., Emmert, A., Bremmer, F., Strecker, J., Holland, R., Hinterthaner, M., Corso, J., Wagner, S., Küffer, S., Sebastian, M., Bergmann, L., Danner, B., Schöndube, F. A., Serve, H., Urlaub, H., & Oellerich, T. (2014). Comprehensive quantitative proteomic profiling of lung cancers reveals novel biomarkers and potential drug targets. Cancer Research, 74(Supplement S): 2487. [PubMan] : Oellerich, T., Mohr, S., Beck, J., Bohnenberger, H., Doebele, C., Corso, J., Burg, G., Schütz, E., Urlaub, H., & Serve, H. (2014). Syk and Btk transduce survival and proliferation-inducing signals by activating distinct signaling pathways and transcriptional programs in AML cells. Blood, 124(21). [PubMan] : Oellerich, T., Oellerich, M. F., Engelke, M., Münch, S., Mohr, S., Nimz, M., Hsiao, H. H., Corso, J., Zhang, J., Bohnenberger, H., Berg, T., Rieger, M. A., Wienands, J., Bug, G., Brandts, C., Urlaub, H., & Serve, H. (2013). β2 integrin–derived signals induce cell survival and proliferation of AML blasts by activating a Syk/STAT signaling axis. Blood, 121(19), 3889-3899. doi:10.1182/blood-2012-09-457887. [PubMan] : Urlaub, H., Corso, J., Oellerich, T., & Serve, H. (2013). Quantitative mass spectrometry: Elucidation of the transduction cascade triggered by B cell receptors. GIT Laboratory Journal Europe, 17(1-2), 22-24. [PubMan] : Oellerich, T., Oellerich, M. F., Engelke, M., Münch, S., Mohr, S., Nimz, M., Hsiao, H. H., Corso, J., Zhang, J., Bohnenberger, H., Berg, T., Rieger, M. A., Wienands, J., Bug, G., Brandts, C., Urlaub, H., & Serve, H. (2013). β2 integrin–derived signals induce cell survival and proliferation of AML blasts by activating a Syk/STAT signaling axis. Cancer Research, 73(Supplement 1): 1107. [PubMan]