Researcher Portfolio

 
   

Smola, Alexander J

Department Empirical Inference, Max Planck Institute for Biological Cybernetics, Max Planck Society, Dept. Empirical Inference, Max Planck Institute for Intelligent Systems, Max Planck Society, Max Planck Institute for Biological Cybernetics, Max Planck Society  

 

Researcher Profile

 
Position: Department Empirical Inference, Max Planck Institute for Biological Cybernetics, Max Planck Society
Position: Max Planck Institute for Biological Cybernetics, Max Planck Society
Position: Dept. Empirical Inference, Max Planck Institute for Intelligent Systems, Max Planck Society
Additional IDs: MPIKYB: smola
Researcher ID: https://pure.mpg.de/cone/persons/resource/persons84953

External references

 
WorldCat Search for Smola, Alexander J
Google Scholar Search for Smola, Alexander J

Publications

 
 
 : Jin, Y.-J., Liang, G., Li, R., Wang, S., Alnouri, M. W., Bentsen, M., Kuenne, C., Guenther, S., Yan, Y., Li, Y., Wettschureck, N., & Offermanns, S. (2025). Phosphorylation of endothelial histone H3.3 serine 31 by PKN1 links flow-induced signaling to proatherogenic gene expression. NATURE CARDIOVASCULAR RESEARCH, 4(2). doi:10.1038/s44161-024-00593-y. [PubMan] : Liang, G., Wang, S., Shao, J., Jin, Y.-J., Xu, L., Yan, Y., Guenther, S., Wang, L., & Offermanns, S. (2022). Tenascin-X Mediates Flow-Induced Suppression of EndMT and Atherosclerosis. CIRCULATION RESEARCH, 130(11), 1647-1659. doi:10.1161/CIRCRESAHA.121.320694. [PubMan] : Jin, Y.-J., Chennupati, R., Li, R., Liang, G., Wang, S., Iring, A., Graumann, J., Wettschureck, N., & Offermanns, S. (2021). Protein kinase N2 mediates flow-induced endothelial NOS activation and vascular tone regulation. JOURNAL OF CLINICAL INVESTIGATION, 131(21): e145734. doi:10.1172/JCI145734. [PubMan] : Nakayama, A., Albarran-Juarez, J., Liang, G., Roquid, K. A., Iring, A., Tonack, S., Chen, M., Mueller, O. J., Weinstein, L. S., & Offermanns, S. (2020). Disturbed flow-induced G(s)-mediated signaling protects against endothelial inflammation and atherosclerosis. JCI INSIGHT, 5(23): e140485. doi:10.1172/jci.insight.140485. [PubMan]