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  The simultaneous loss of Arx and Pax4 genes promotes a somatostatin-producing cell fate specification at the expense of the alpha- and beta-cell lineages in the mouse endocrine pancreas

Collombat, P., Hecksher-Soerensen, J., Broccoli, V., Krull, J., Ponte, I., Mundiger, T., et al. (2005). The simultaneous loss of Arx and Pax4 genes promotes a somatostatin-producing cell fate specification at the expense of the alpha- and beta-cell lineages in the mouse endocrine pancreas. Development, 132(13), 2969-2980. Retrieved from http://dev.biologists.org/cgi/content/full/132/13/2969.

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Item Permalink: https://hdl.handle.net/11858/00-001M-0000-0012-E8A3-6 Version Permalink: https://hdl.handle.net/11858/00-001M-0000-002A-0F23-6
Genre: Journal Article

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Collombat, P.1, Author           
Hecksher-Soerensen, J., Author
Broccoli, V., Author
Krull, J.1, Author           
Ponte, I., Author
Mundiger, T., Author
Smith, J., Author
Gruss, P.2, Author
Serup, P., Author
Mansouri, A.1, Author           
Affiliations:
1Research Group of Molecular Cell Differentiation, MPI for biophysical chemistry, Max Planck Society, ou_578588              
2Max Planck Society, ou_persistent13              

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Free keywords: endocrine pancreas development; Arx; Pax4; mouse; hyperglycaemia; fate specification
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Language(s): eng - English
 Dates: 2005-07
 Publication Status: Issued
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 Rev. Type: Peer
 Identifiers: eDoc: 256002
URI: http://dev.biologists.org/cgi/content/full/132/13/2969
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Title: Development
Source Genre: Journal
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Pages: - Volume / Issue: 132 (13) Sequence Number: - Start / End Page: 2969 - 2980 Identifier: -