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  Heterozygosity for the mutated X-chromosome-linked L1 cell adhesion molecule gene leads to increased numbers of neurons and enhanced metabolism in the forebrain of female carrier mice

Schmid, J. S., Bernreuther, C., Nikonenko, A. G., Ling, Z., Mies, G., Hossmann, K.-A., et al. (2013). Heterozygosity for the mutated X-chromosome-linked L1 cell adhesion molecule gene leads to increased numbers of neurons and enhanced metabolism in the forebrain of female carrier mice. Brain Structure and Function, 218(6), 1375-1390. doi:10.1007/s00429-012-0463-9.

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 Creators:
Schmid, Janinne Sylvie1, Author
Bernreuther, Christian1, Author
Nikonenko, Alexander G.1, Author
Ling, Zhang1, Author
Mies, Günter2, Author           
Hossmann, Konstantin-Alexander3, Author           
Jakovcevski, Igor1, Author
Schachner, Melitta1, Author
Affiliations:
1a Center for Molecular Neurobiology, University Medical Center Hamburg-Eppendorf, Martinistraße 52, Hamburg, 20246, Germany b Institute of Neuropathology, University Medical Center Hamburg-Eppendorf, Martinistraße 52, Hamburg, 20246, Germany c Department of Cytology, Bogomoletz Institute of Physiology, Bogomoletz Street 4, Kiev, 01024, Ukraine d W. M. Keck Center for Collaborative Neuroscience and Department of Cell Biology and Neuroscience, Rutgers University, 604 Allison Road, Piscataway, 08854, United States e Center for Neuroscience, Shantou University Medical College, Shantou, 515041, China, ou_persistent22              
2Multimodal Imaging of Brain Metabolism, Research Groups, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society, ou_2149667              
3Konstantin-Alexander Hossmann, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society, ou_2149648              

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 Abstract: © 2012 Springer-Verlag Berlin Heidelberg.

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Language(s): eng - English
 Dates: 2013
 Publication Status: Issued
 Pages: -
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 Rev. Type: Peer
 Identifiers: eDoc: 628937
DOI: 10.1007/s00429-012-0463-9
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Title: Brain Structure and Function
Source Genre: Journal
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Pages: - Volume / Issue: 218 (6) Sequence Number: - Start / End Page: 1375 - 1390 Identifier: ISSN: 18632653