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  CX3CR1 disruption differentially influences dopaminergic neuron degeneration in Parkinsonian mice depending on the neurotoxin and route of administration

Tristão, F. S. M., Lazzarini, M., Martin, S., Amar, M., Stühmer, W., Kirchhoff, F., et al. (2015). CX3CR1 disruption differentially influences dopaminergic neuron degeneration in Parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity research, 10.1007/s12640-015-9557-5. doi:10.1007/s12640-015-9557-5.

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 Creators:
Tristão, Fabrine Sales Massafera, Author
Lazzarini, Marcio1, Author           
Martin, Sabine1, Author           
Amar, Majid, Author
Stühmer, Walter1, Author           
Kirchhoff, Frank, Author
Caldi Gomes, Lucas Araújo1, Author           
Lanfumey, Laurance, Author
Prediger, Rui D., Author
Sepulveda, Julia E., Author
Del-Bel, Elaine A., Author
Raisman-Vozari, Rita, Author
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1Molecular biology of neuronal signals, Max Planck Institute of Experimental Medicine, Max Planck Society, ou_2173656              

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Free keywords: Parkinson’s disease; MPTP; 6-OHDA; Fractalkine (CX3CL1); Fractalkine receptor (CX3CR1); Neurodegeneration; Neuroinflammation
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 Dates: 2015-08-182015-09-24
 Publication Status: Published online
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 Rev. Type: Peer
 Identifiers: DOI: 10.1007/s12640-015-9557-5
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Title: Neurotoxicity research
Source Genre: Journal
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Pages: - Volume / Issue: - Sequence Number: 10.1007/s12640-015-9557-5 Start / End Page: - Identifier: -