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  Stimulus dependent laminar differences in functional CBF in monkey V1

Goense, J., Merkle, H., & Logothetis, N. (2012). Stimulus dependent laminar differences in functional CBF in monkey V1. Poster presented at 20th Annual Meeting and Exhibition of the International Society for Magnetic Resonance in Medicine (ISMRM 2012), Melbourne, Australia.

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Item Permalink: http://hdl.handle.net/11858/00-001M-0000-0013-B7BE-6 Version Permalink: http://hdl.handle.net/21.11116/0000-0001-9EE4-C
Genre: Poster

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 Creators:
Goense, J1, 2, Author              
Merkle, H, Author              
Logothetis, NK1, 2, Author              
Affiliations:
1Department Physiology of Cognitive Processes, Max Planck Institute for Biological Cybernetics, Max Planck Society, ou_1497798              
2Max Planck Institute for Biological Cybernetics, Max Planck Society, ou_1497794              

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 Abstract: The relative contributions of excitation and inhibition to fMRI responses remain unknown. In principle, inhibition may increase or decrease fMRI signals depending on local circuitry. Negative BOLD signals and CBF decreases were shown for ring stimuli in primary visual cortex (V1). High-resolution fMRI can exploit the functional segregation in V1 to reveal differences between excitatory and inhibitory responses, including layer-specific differences. We measured high-resolution BOLD, CBV and CBF in macaque V1 and found laminar differences in the positive and negative fCBF responses, suggesting different neurovascular coupling mechanisms depending on the location within the cortical sheet.

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 Dates: 2012-05
 Publication Status: Published in print
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 Identifiers: BibTex Citekey: GoenseML2012
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Title: 20th Annual Meeting and Exhibition of the International Society for Magnetic Resonance in Medicine (ISMRM 2012)
Place of Event: Melbourne, Australia
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Title: 20th Annual Meeting and Exhibition of the International Society for Magnetic Resonance in Medicine (ISMRM 2012)
Source Genre: Proceedings
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Pages: - Volume / Issue: - Sequence Number: 0718 Start / End Page: - Identifier: -