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  The Negative BOLD Response in Monkey V1 is Associated with Decreases in Neuronal Activity

Shmuel, A., Augath, M., Oeltermann, A., Pauls, J., & Logothetis, N. (2003). The Negative BOLD Response in Monkey V1 is Associated with Decreases in Neuronal Activity. In 11th Scientific Meeting of the International Society of Magnetic Resonance in Medicine (ISMRM 2003) (pp. 51).

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Item Permalink: http://hdl.handle.net/11858/00-001M-0000-0013-DC39-4 Version Permalink: http://hdl.handle.net/21.11116/0000-0005-7770-7
Genre: Meeting Abstract

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 Creators:
Shmuel, A1, 2, Author              
Augath, MA1, 2, Author              
Oeltermann, A1, 2, Author              
Pauls, J1, 2, Author              
Logothetis, NK1, 2, Author              
Affiliations:
1Department Physiology of Cognitive Processes, Max Planck Institute for Biological Cybernetics, Max Planck Society, ou_1497798              
2Max Planck Institute for Biological Cybernetics, Max Planck Society, ou_1497794              

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 Abstract: This study aimed at revealing the neuronal correlates of the negative BOLD response (NBR). Electrical recordings were obtained from NBR regions in monkey V1 simultaneously with fMRI. The NBR was associated with a reduction in neuronal activity, both in the Multi-Unit-Activity and the Local-Field-Potential domains. The onset of the decrease in neuronal signal preceded the corresponding onset of the NBR, indicating that the origin of the NBR was a decrease in neuronal activity that triggered a reduction in CBF, rather than decreased neuronal activity caused by reduced CBF.

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 Dates: 2003-07
 Publication Status: Published in print
 Pages: -
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 Rev. Method: -
 Identifiers: BibTex Citekey: 2407
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Title: 11th Scientific Meeting of the International Society of Magnetic Resonance in Medicine (ISMRM 2003)
Place of Event: Toronto, Canada
Start-/End Date: 2003-07-10 - 2003-07-16

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Title: 11th Scientific Meeting of the International Society of Magnetic Resonance in Medicine (ISMRM 2003)
Source Genre: Proceedings
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Publ. Info: -
Pages: - Volume / Issue: - Sequence Number: 211 Start / End Page: 51 Identifier: ISSN: 1545-4428