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  Reply to Christ et al.: LQT1 and JLNS phenotypes in hiPSC-derived cardiomyocytes are due to KCNQ1 mutations

Greber, B., Verkerk, A. O., Seebohm, G., Mummery, C. L., & Bellin, M. (2015). Reply to Christ et al.: LQT1 and JLNS phenotypes in hiPSC-derived cardiomyocytes are due to KCNQ1 mutations. Proceedings of the National Academy of Sciences of the United States of America, 112(16), E1969:1-E1969:1. doi:10.1073/pnas.1503762112.

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 Creators:
Greber, Boris1, Author           
Verkerk, Arie O.2, Author
Seebohm, Guiscard2, Author
Mummery, Christine L.2, Author
Bellin, Milena2, Author
Affiliations:
1Chemical Genomics Center of the MPS, Max Planck Institute of Molecular Physiology, Max Planck Society, ou_1753292              
2External Organizations, ou_persistent22              

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Language(s): eng - English
 Dates: 2015-03-20
 Publication Status: Issued
 Pages: -
 Publishing info: -
 Table of Contents: -
 Rev. Type: Peer
 Identifiers: DOI: 10.1073/pnas.1503762112
 Degree: -

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Title: Proceedings of the National Academy of Sciences of the United States of America
  Other : Proc. Natl. Acad. Sci. U. S. A.
Source Genre: Journal
 Creator(s):
Affiliations:
Publ. Info: National Academy of Sciences
Pages: - Volume / Issue: 112 (16) Sequence Number: - Start / End Page: E1969:1 - E1969:1 Identifier: ISSN: 0027-8424
CoNE: https://pure.mpg.de/cone/journals/resource/954925427230