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  Insulin receptor signaling mediates APP processing and β-amyloid accumulation without altering survival in a transgenic mouse model of Alzheimer's disease

Stöhr, O., Schilbach, K., Moll, L., Hettich, M. M., Freude, S., Wunderlich, F. T., et al. (2013). Insulin receptor signaling mediates APP processing and β-amyloid accumulation without altering survival in a transgenic mouse model of Alzheimer's disease. AGE, 35(1), 83-101. doi:10.1007/s11357-011-9333-2.

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 Creators:
Stöhr, Oliver1, Author
Schilbach, Katharina1, Author
Moll, Lorna1, Author
Hettich, Moritz M.1, Author
Freude, Susanna1, Author
Wunderlich, F. Thomas2, Author           
Ernst, Marianne1, Author
Zemva, Johanna1, Author
Brüning, Jens C.3, Author           
Krone, Wilhelm1, Author
Udelhoven, Michael1, Author
Schubert, Markus1, Author
Affiliations:
1a Center for Endocrinology, Diabetes and Preventive Medicine (CEDP), Center for Molecular Medicine Col, University of Cologne, Kerpener Str 62, Cologne, 50931, Germany b Cologne excellence cluster in Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Cologne, Germany c Molecular and Cellular Cognition Lab, German Centre for Neurodegenerative Diseases (DZNE), Bonn, 53175, Germany d Department of Mouse Genetics and Metabolism, Institute for Genetics University of Cologne, Zülpicher Strasse 47, Cologne, 50674, Germany e Center for Endocrinology, Diabetes and Preventive Medicine, University of Cologne, CMMC building 66, 5.012, Robert-Koch-Str. 21, Cologne, 50931, Germany, ou_persistent22              
2Obesity and Cancer, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society, ou_2149645              
3Neuronal Control of Metabolism, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society, ou_2149644              

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 Abstract: © 2011 American Aging Association.

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Language(s): eng - English
 Dates: 2013
 Publication Status: Published in print
 Pages: -
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 Table of Contents: -
 Rev. Type: Peer
 Identifiers: eDoc: 573598
DOI: 10.1007/s11357-011-9333-2
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Title: AGE
Source Genre: Journal
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Pages: - Volume / Issue: 35 (1) Sequence Number: - Start / End Page: 83 - 101 Identifier: ISSN: 01619152