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  Loss of UCP2 attenuates mitochondrial dysfunction without altering ROS production and uncoupling activity

Kukat, A., Dogan, S. A., Edgar, D., Mourier, A., Jacoby, C., Maiti, P., et al. (2014). Loss of UCP2 attenuates mitochondrial dysfunction without altering ROS production and uncoupling activity. PLoS Genetics, 10(6): e1004385. doi:10.1371/journal.pgen.1004385.

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 Creators:
Kukat, Alexandra1, Author
Dogan, Sukru Anil1, Author
Edgar, Daniel1, Author
Mourier, Arnaud1, Author
Jacoby, Christoph1, Author
Maiti, Priyanka1, Author
Mauer, Jan2, Author           
Becker, Christina1, Author
Senft, Katharina1, Author
Wibom, Rolf1, Author
Kudin, Alexei P.1, Author
Hultenby, Kjell1, Author
Flögel, Ulrich1, Author
Rosenkranz, Stephan1, Author
Ricquier, Daniel1, Author
Kunz, Wolfram S.1, Author
Trifunovic, Aleksandra1, Author
Affiliations:
1a Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), Institute for Mitochondrial Diseases and Aging, University of Cologne, Cologne, Germany b Department of Laboratory Medicine, Karolinska Institutet, Stockholm, Sweden c Max Planck Institute for Biology of Aging, Cologne, Germany d Department of Cardiovascular Physiology, Heinrich-Heine-University, Düsseldorf, Germany e Department of Epileptology, University of Bonn, Bonn, Germany f Department III of Internal Medicine, University of Cologne, Cologne, Germany g Cologne Cardiovascular Research Center (CCRC) and Center for Molecular Medicine Cologne, University of Cologne, Cologne, Germany h University Paris Descartes, CNRS FRE3210, Paris, France, ou_persistent22              
2Neuronal Control of Metabolism, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society, ou_2149644              

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 Abstract: © 2014 Kukat et al.

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Language(s): eng - English
 Dates: 2014
 Publication Status: Issued
 Pages: -
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 Table of Contents: -
 Rev. Type: Peer
 Identifiers: eDoc: 695778
DOI: 10.1371/journal.pgen.1004385
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Title: PLoS Genetics
Source Genre: Journal
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Pages: - Volume / Issue: 10 (6) Sequence Number: e1004385 Start / End Page: - Identifier: ISSN: 15537404