Signaling through the adaptor molecule MyD88 in CD4(+) T cells is required to 
overcome suppression by regulatory T cells

Schenten, Dominik; Nish, Simone A.; Yu, Shuang; Yan, Xiting; Lee, Heung Kyu; Brodsky, Igor; Pasman, Lesley; Yordy, Brian; Wunderlich, F. Thomas; Brüning, Jens C.; Zhao, Hongyu; Medzhitov, Ruslan

doi:10.1016/j.immuni.2013.10.023

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Signaling through the adaptor molecule MyD88 in CD4(+) T cells is required to overcome suppression by regulatory T cells

Schenten, D., Nish, S. A., Yu, S., Yan, X., Lee, H. K., Brodsky, I., et al. (2014). Signaling through the adaptor molecule MyD88 in CD4(+) T cells is required to overcome suppression by regulatory T cells. Immunity, 40(1), 78-90. doi:10.1016/j.immuni.2013.10.023.

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Item Permalink: https://hdl.handle.net/11858/00-001M-0000-0026-D905-4 Version Permalink: https://hdl.handle.net/11858/00-001M-0000-0026-D906-2

Genre: Journal Article

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Creators:
Schenten, Dominik¹, Author
Nish, Simone A.¹, Author
Yu, Shuang¹, Author
Yan, Xiting¹, Author
Lee, Heung Kyu¹, Author
Brodsky, Igor¹, Author
Pasman, Lesley¹, Author
Yordy, Brian¹, Author
Wunderlich, F. Thomas², Author
Brüning, Jens C.³, Author
Zhao, Hongyu¹, Author
Medzhitov, Ruslan¹, Author

Affiliations:
1[ 1 ] Yale Univ, Dept Immunobiol, New Haven, CT 06519 USA [ 2 ] Yale Univ, Sch Med, Howard Hughes Med Inst, New Haven, CT 06519 USA [ 3 ] Yale Univ, Sch Publ Hlth, Dept Biostat, New Haven, CT 06520 USA [ 4 ] Univ Cologne, Ctr Endocrinol Diabet & Prevent Med CEDP, CMMC, Inst Genet, D-50931 Cologne, Germany, ou_persistent22
2Obesity and Cancer, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society, ou_2149645
3Neuronal Control of Metabolism, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society, ou_2149644

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Language(s): eng - English

Dates: Date issued: 2014

Publication Status: Issued

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Rev. Type: Peer

Identifiers: eDoc: 695177
DOI: 10.1016/j.immuni.2013.10.023

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Title: Immunity

Source Genre: Journal

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Pages: - Volume / Issue: 40 (1) Sequence Number: - Start / End Page: 78 - 90 Identifier: ISSN: 1074-7613