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  Axonal degeneration in an Alzheimer mouse model is PS1 gene dose dependent and linked to intraneuronal Aβ accumulation

Christensen, D. Z., Huettenrauch, M., Mitkovski, M., Pradier, L., & Wirths, O. (2014). Axonal degeneration in an Alzheimer mouse model is PS1 gene dose dependent and linked to intraneuronal Aβ accumulation. Frontiers in Aging Neuroscience, 6: 139. doi:10.3389/fnagi.2014.00139.

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Item Permalink: https://hdl.handle.net/11858/00-001M-0000-002A-F191-C Version Permalink: https://hdl.handle.net/11858/00-001M-0000-002A-F192-A
Genre: Journal Article

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 Creators:
Christensen, Ditte Z., Author
Huettenrauch, Melanie, Author
Mitkovski, Miso1, Author           
Pradier, Laurent, Author
Wirths, Oliver, Author
Affiliations:
1Light microscopy facility, Wiss. Servicegruppen, Max Planck Institute of Experimental Medicine, Max Planck Society, ou_2173672              

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Free keywords: Amyloid; Presenilin; Axonal degeneration; Axonopathy; Intraneuronal Abeta; Transgenic mice; Alzheimer; Axonal transport
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Language(s): eng - English
 Dates: 2014-06-27
 Publication Status: Issued
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 Table of Contents: -
 Rev. Type: Peer
 Identifiers: eDoc: 695941
DOI: 10.3389/fnagi.2014.00139
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Title: Frontiers in Aging Neuroscience
Source Genre: Journal
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Pages: - Volume / Issue: 6 Sequence Number: 139 Start / End Page: - Identifier: -