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  CX3CR1 disruption differentially influences dopaminergic neuron degeneration in Parkinsonian mice depending on the neurotoxin and route of administration

Tristão, F. S. M., Lazzarini, M., Martin, S., Amar, M., Stühmer, W., Kirchhoff, F., et al. (2016). CX3CR1 disruption differentially influences dopaminergic neuron degeneration in Parkinsonian mice depending on the neurotoxin and route of administration. Neurotoxicity research, 29(3), 364-380. doi:10.1007/s12640-015-9557-5.

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 Creators:
Tristão, Fabrine Sales Massafera, Author
Lazzarini, Marcio1, Author              
Martin, Sabine1, Author              
Amar, Majid, Author
Stühmer, Walter1, Author              
Kirchhoff, Frank, Author
Caldi Gomes, Lucas Araújo1, Author              
Lanfumey, Laurance, Author
Prediger, Rui D., Author
Sepulveda, Julia E., Author
Del-Bel, Elaine A., Author
Raisman-Vozari, Rita, Author
Affiliations:
1Molecular biology of neuronal signals, Max Planck Institute of Experimental Medicine, Max Planck Society, ou_2173656              

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Free keywords: Parkinson’s disease; MPTP; 6-OHDA; Fractalkine (CX3CL1); Fractalkine receptor (CX3CR1); Neurodegeneration; Neuroinflammation
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Language(s): eng - English
 Dates: 2015-08-182015-09-242016-04
 Publication Status: Published in print
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 Rev. Type: Peer
 Identifiers: DOI: 10.1007/s12640-015-9557-5
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Title: Neurotoxicity research
Source Genre: Journal
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Pages: - Volume / Issue: 29 (3) Sequence Number: - Start / End Page: 364 - 380 Identifier: -