Tau mutant A152T, a risk factor for FTD/PSP, induces neuronal dysfunction and 
reduced lifespan independently of aggregation in a C. elegans Tauopathy model

Pir, Gulam Jeelani; Choudhary, Bikash; Mandelkow, Eckhard; Mandelkow, Eva-Maria

doi:10.1186/s13024-016-0096-1

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Tau mutant A152T, a risk factor for FTD/PSP, induces neuronal dysfunction and reduced lifespan independently of aggregation in a C. elegans Tauopathy model

Pir, G. J., Choudhary, B., Mandelkow, E., & Mandelkow, E.-M. (2016). Tau mutant A152T, a risk factor for FTD/PSP, induces neuronal dysfunction and reduced lifespan independently of aggregation in a C. elegans Tauopathy model. Molecular Neurodegeneration, 11(1): 96. doi:10.1186/s13024-016-0096-1.

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Datensatz-Permalink: https://hdl.handle.net/11858/00-001M-0000-002A-70D3-9 Versions-Permalink: https://hdl.handle.net/11858/00-001M-0000-002A-70D4-7

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Urheber:
Pir, Gulam Jeelani¹, Autor
Choudhary, Bikash, Autor
Mandelkow, Eckhard², Autor
Mandelkow, Eva-Maria², Autor

Affiliations:
1Neuronal Control of Metabolism, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society, ou_2149644
2Eckhard Mandelkow, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society, ou_2149647