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  Abnormal contractility in human heart myofibrils from patients with dilated cardiomyopathy due to mutations in TTN and contractile protein genes

Vikhorev, P. G., Smoktunowicz, N., Munster, A. B., Copeland, O., Kostin, S., Montgiraud, C., et al. (2017). Abnormal contractility in human heart myofibrils from patients with dilated cardiomyopathy due to mutations in TTN and contractile protein genes. SCIENTIFIC REPORTS, 7: 14829. doi:10.1038/s41598-017-13675-8.

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 Creators:
Vikhorev, Petr G., Author
Smoktunowicz, Natalia, Author
Munster, Alex B., Author
Copeland, O'Neal, Author
Kostin, Sawa1, Author           
Montgiraud, Cecile, Author
Messer, Andrew E., Author
Toliat, Mohammad R., Author
Li, Amy, Author
dos Remedios, Cristobal G., Author
Lal, Sean, Author
Blair, Cheavar A., Author
Campbell, Kenneth S., Author
Guglin, Maya, Author
Knoll, Ralph, Author
Marston, Steven B., Author
Affiliations:
1Electron Microscopy, Max Planck Institute for Heart and Lung Research, Max Planck Society, ou_2591707              

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Free keywords: TROPONIN-I PHOSPHORYLATION; LENGTH-DEPENDENT ACTIVATION; HUMAN CARDIAC MYOFIBRILS; FRANK-STARLING MECHANISM; TENSION GENERATION; HYPERTROPHIC CARDIOMYOPATHY; CA2+ SENSITIVITY; TITIN; FAILURE; MYOSIN
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 Dates: 2017
 Publication Status: Published online
 Pages: -
 Publishing info: -
 Table of Contents: -
 Rev. Type: -
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Title: SCIENTIFIC REPORTS
Source Genre: Journal
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Pages: - Volume / Issue: 7 Sequence Number: 14829 Start / End Page: - Identifier: ISSN: 2045-2322