20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner 
through FFAR1

Tunaru, Sorin; Bonnavion, Remy; Brandenburger, Isabell; Preussner, Jens; Thomas, Dominique; Scholich, Klaus; Offermanns, Stefan

doi:10.1038/s41467-017-02539-4

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20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner through FFAR1

Tunaru, S., Bonnavion, R., Brandenburger, I., Preussner, J., Thomas, D., Scholich, K., et al. (2018). 20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner through FFAR1. NATURE COMMUNICATIONS, 9: 177. doi:10.1038/s41467-017-02539-4.

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Item Permalink: https://hdl.handle.net/21.11116/0000-0004-5E1D-4 Version Permalink: https://hdl.handle.net/21.11116/0000-0007-45BD-7

Genre: Journal Article

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Creators:
Tunaru, Sorin¹, Author
Bonnavion, Remy¹, Author
Brandenburger, Isabell¹, Author
Preussner, Jens², Author
Thomas, Dominique, Author
Scholich, Klaus, Author
Offermanns, Stefan¹, Author

Affiliations:
1Pharmacology, Max Planck Institute for Heart and Lung Research, Max Planck Society, ou_2591696
2Bioinformatics, Max Planck Institute for Heart and Lung Research, Max Planck Society, ou_2591704

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Dates: Published Online: 2018

Publication Status: Published online

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Identifiers: ISI: 000419949200004
DOI: 10.1038/s41467-017-02539-4

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Title: NATURE COMMUNICATIONS

Source Genre: Journal

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Pages: - Volume / Issue: 9 Sequence Number: 177 Start / End Page: - Identifier: ISSN: 2041-1723