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  Mice lacking the mitochondrial exonuclease MGME1 accumulate mtDNA deletions without developing progeria

Matic, S., Jiang, M., Nicholls, T. J., Uhler, J. P., Dirksen-Schwanenland, C., Polosa, P. L., et al. (2018). Mice lacking the mitochondrial exonuclease MGME1 accumulate mtDNA deletions without developing progeria. Nat Commun, 9(1), 1202. doi:10.1038/s41467-018-03552-x.

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Basic (Discarded)

Date of Discard: 2022-10-31
Comment: doppelt
 Creators:
Matic, S.1           
Jiang, M.1           
Nicholls, T. J.
Uhler, J. P.
Dirksen-Schwanenland, C.1           
Polosa, P. L.
Simard, Marie-Lune1           
Li, X.2           
Atanassov, Ilian2           
Rackham, O.
Filipovska, A.
Stewart, J.3           
Falkenberg, M.
Larsson, N.G.1           
Milenkovic, D.1           
Affiliations:
1Department Larsson - Mitochondrial Biology, Max Planck Institute for Biology of Ageing, Max Planck Society, ou_1942286              
2Proteomics, Core Facilities, Max Planck Institute for Biology of Ageing, Max Planck Society, ou_1942305              
3Stewart – Mitochondrial Mutations and Genome Co-evolution, Research Groups, Max Planck Institute for Biology of Ageing, Max Planck Society, ou_1942301              
 Dates: 2018-03-232018
Files: 0 Files
Locators: 1 Locator
version ID: item_3151554_3
Item State: Discarded
Name of Context: Publications of the MPI for Biology of Ageing, Affiliated to: Max Planck Institute for Biology of Ageing