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Abstract:
Direct or indirect stimulation of brain activity through brain machine interfaces holds the promise of triggering the enduring network modifications necessary to functional recovery. Possibly daunting at first, this aim appears more reachable with regard to the dramatic plastic changes occurring in healthy brains on a daily basis. In light of recent results, I will discuss the putative mechanisms exploited by the mammalian brain to control plasticity during sleep. I will then elaborate on how such mechanisms, originally promoting long-term memory and homeostasis, can be possibly leveraged for recovery.
