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  Mitofusin 2 builds a bridge between ER and mitochondria

Merkwirth, C., & Langer, T. (2008). Mitofusin 2 builds a bridge between ER and mitochondria. Cell, 135(7), 1165-7. doi:10.1016/j.cell.2008.12.005.

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Item Permalink: https://hdl.handle.net/21.11116/0000-000B-80FB-9 Version Permalink: https://hdl.handle.net/21.11116/0000-000B-80FC-8
Genre: Journal Article

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https://www.ncbi.nlm.nih.gov/pubmed/19109886 (Any fulltext)
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Merkwirth, C., Author
Langer, T.1, Author           
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1Department Langer - Mitochondrial Proteostasis, Max Planck Institute for Biology of Ageing, Max Planck Society, ou_3393994              

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Free keywords: Animals Axonal Transport Cell Line Endoplasmic Reticulum/*metabolism GTP Phosphohydrolases/*metabolism Mice Mitochondria/*metabolism Purkinje Cells/metabolism
 Abstract: Mutations in mitofusin 2 (MFN2), a dynamin-like GTPase required for mitochondrial fusion, cause the peripheral neuropathy Charcot-Marie-Tooth type 2A. In a recent report in Nature, de Brito and Scorrano (2008) demonstrate a new function of MFN2-tethering the endoplasmic reticulum and mitochondria to control the efficiency of mitochondrial uptake of Ca2+ ions.

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 Dates: 2008-12-262008-12-27
 Publication Status: Issued
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 Identifiers: Other: 19109886
DOI: 10.1016/j.cell.2008.12.005
ISSN: 1097-4172 (Electronic)0092-8674 (Linking)
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Title: Cell
Source Genre: Journal
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Pages: - Volume / Issue: 135 (7) Sequence Number: - Start / End Page: 1165 - 7 Identifier: -