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Mutation in the transcriptional regulator PhoP contributes to avirulence of Mycobacterium tuberculosis H37Ra strain

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Lee,  Jong Seok
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Krause,  Roland
Department of Cellular Microbiology, Max Planck Institute for Infection Biology;
Dept. of Computational Molecular Biology (Head: Martin Vingron), Max Planck Institute for Molecular Genetics, Max Planck Society;

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Schreiber,  Jörg
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Mollenkopf,  Hans J.
Core Facilities / Microarray, Max Planck Institute for Infection Biology, Max Planck Society;

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Kowall,  Jane
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Stein,  Robert
Core Facilities / Proteinanalysis, Max Planck Institute for Infection Biology, Max Planck Society;

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Patron,  Juan Pablo
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Jorg,  Sabine
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Kaufmann,  Stefan H. E.
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Citation

Lee, J. S., Krause, R., Schreiber, J., Mollenkopf, H. J., Kowall, J., Stein, R., et al. (2008). Mutation in the transcriptional regulator PhoP contributes to avirulence of Mycobacterium tuberculosis H37Ra strain. Cell Host & Microbe, 3(2), 97-103.


Cite as: http://hdl.handle.net/11858/00-001M-0000-000E-C1F3-7
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