N-truncated amyloid beta (A beta) 4-42 forms stable aggregates and induces 
acute and long-lasting behavioral deficits.

Bouter, I.; Dittrich, K.; Wittnam, J. L.; Rezaei-Ghaleh, N.; Pillot, T.; Papot-Coutourier, S.; Lefebvre, T.; Sprenger, F.; Wirths, O.; Zweckstetter, M.; Bayer, T. A.

doi:10.1007/s00401-013-1129-2

Item

ITEM ACTIONSEXPORT

Add to Basket

Local TagsRelease HistoryDetailsSummary

Released

Journal Article

N-truncated amyloid beta (A beta) 4-42 forms stable aggregates and induces acute and long-lasting behavioral deficits.

MPS-Authors

/persons/resource/persons15124

Rezaei-Ghaleh, N.
Research Group of Protein Structure Determination using NMR, MPI for biophysical chemistry, Max Planck Society;

/persons/resource/persons16093

Zweckstetter, M.
Research Group of Protein Structure Determination using NMR, MPI for biophysical chemistry, Max Planck Society;

External Resource

http://link.springer.com/content/pdf/10.1007%2Fs00401-013-1129-2.pdf
(Publisher version)

Fulltext (restricted access)

There are currently no full texts shared for your IP range.

Fulltext (public)

1834518.pdf
(Publisher version), 2MB

Supplementary Material (public)

There is no public supplementary material available

Citation

Bouter, I., Dittrich, K., Wittnam, J. L., Rezaei-Ghaleh, N., Pillot, T., Papot-Coutourier, S., et al. (2013). N-truncated amyloid beta (A beta) 4-42 forms stable aggregates and induces acute and long-lasting behavioral deficits. Acta Neuropathologica, 126(2), 189-205. doi:10.1007/s00401-013-1129-2.

Cite as: https://hdl.handle.net/11858/00-001M-0000-0014-470D-8

Abstract

There is no abstract available