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Reply to Christ et al.: LQT1 and JLNS phenotypes in hiPSC-derived cardiomyocytes are due to KCNQ1 mutations

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Greber,  Boris
Chemical Genomics Center of the MPS, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Greber, B., Verkerk, A. O., Seebohm, G., Mummery, C. L., & Bellin, M. (2015). Reply to Christ et al.: LQT1 and JLNS phenotypes in hiPSC-derived cardiomyocytes are due to KCNQ1 mutations. Proceedings of the National Academy of Sciences of the United States of America, 112(16), E1969:1-E1969:1. doi:10.1073/pnas.1503762112.


Cite as: http://hdl.handle.net/11858/00-001M-0000-0025-B9CF-6
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