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The antiparkinsonian effects of deep brain stimulation in the subthalamic nucleus are not mediated by an increase of tonic striatal dopamine receptor stimulation: evidence from serial [11C]raclopride-PET in parkinsonian humans

MPS-Authors

Hilker,  Rüdiger
Max Planck Society;

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Herholz,  Karl
Wolf-Dieter Heiss, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Ghaemi,  Mehran
Wolf-Dieter Heiss, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Wienhard,  Klaus
Wolf-Dieter Heiss, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Heiss,  Wolf-Dieter
Wolf-Dieter Heiss, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Citation

Hilker, R., Herholz, K., Schuster, A., Voges, J., Ghaemi, M., Sturm, V., et al. (2002). The antiparkinsonian effects of deep brain stimulation in the subthalamic nucleus are not mediated by an increase of tonic striatal dopamine receptor stimulation: evidence from serial [11C]raclopride-PET in parkinsonian humans. NeuroImage, 16(3/P2), S63-S63.


Cite as: https://hdl.handle.net/11858/00-001M-0000-0026-D9B9-1
Abstract
Copyright Elsevier Science 2002