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Loss of the neuron-specific F-box protein FBXO41 models an ataxia-like phenotype in mice with neuronal migration defects and degeneration in the cerebellum

MPS-Authors
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Mukherjee,  Chaitali
Cellular molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Holubowska,  Anna
Cellular molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Schwedhelm-Domeyer,  Nicola
Cellular molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Mitkovski,  Miso
Light microscopy facility, Wiss. Servicegruppen, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Lee,  Shih-Ju
Cellular molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Kannan,  Madhuvanthi
Cellular molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Matz,  Annika
Cellular molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Vadhvani,  Mayur
Cellular molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Stegmüller,  Judith
Cellular molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Citation

Mukherjee, C., Holubowska, A., Schwedhelm-Domeyer, N., Mitkovski, M., Lee, S.-J., Kannan, M., et al. (2015). Loss of the neuron-specific F-box protein FBXO41 models an ataxia-like phenotype in mice with neuronal migration defects and degeneration in the cerebellum. Journal of Neuroscience, 35(23), 8701-8717. doi:10.1523/JNEUROSCI.2133-14.2015.


Cite as: https://hdl.handle.net/11858/00-001M-0000-0029-6B47-7
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