Polytherapy with a combination of three repurposed drugs (PXT3003) 
down-regulates Pmp22 over-expression and improves myelination, axonal and 
functional parameters in models of CMT1A neuropathy

Chumakov, Ilya; Milet, Aude; Cholet, Nathalie; Primas, Gwenal; Boucard, Aurlie; Pereira, Yannick; Graudens, Esther; Mandel, Jonas; Laffaire, Julien; Foucquier, Julie; Glibert, Fabrice; Bertrand, Viviane; Nave, Klaus-Armin; Sereda, Michael W.; Vial, Emmanuel; Guedj, Mickal; Hajj, Rodolphe; Nabirotchkin, Serguei; Cohen, Daniel

doi:10.1186/s13023-014-0201-x

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Polytherapy with a combination of three repurposed drugs (PXT3003) down-regulates Pmp22 over-expression and improves myelination, axonal and functional parameters in models of CMT1A neuropathy

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Nave, Klaus-Armin
Neurogenetics, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Sereda, Michael W.
Molecular and translational neurology, Neurogenetics, Max Planck Institute of Experimental Medicine, Max Planck Society;

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引用

Chumakov, I., Milet, A., Cholet, N., Primas, G., Boucard, A., Pereira, Y., Graudens, E., Mandel, J., Laffaire, J., Foucquier, J., Glibert, F., Bertrand, V., Nave, K.-A., Sereda, M. W., Vial, E., Guedj, M., Hajj, R., Nabirotchkin, S., & Cohen, D. (2014). Polytherapy with a combination of three repurposed drugs (PXT3003) down-regulates Pmp22 over-expression and improves myelination, axonal and functional parameters in models of CMT1A neuropathy. Orphanet Journal of Rare Diseases, 9(1):. doi:10.1186/s13023-014-0201-x.

引用: https://hdl.handle.net/11858/00-001M-0000-002A-F732-7

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