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Calmodulin and Munc13 Form a Ca2+ Sensor/Effector Complex that Controls Short-Term Synaptic Plasticity

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Junge,  Harald J.
Molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

/persons/resource/persons182371

Rhee,  Jeong Seop
Neurophysiology of synapse, Molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

/persons/resource/persons182214

Jahn,  Olaf
Molecular neuroendocrinology, Max Planck Institute of Experimental Medicine, Max Planck Society;

/persons/resource/persons182468

Varoqueaux,  Frédérique
Molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

/persons/resource/persons182423

Spiess,  Joachim
Molecular neuroendocrinology, Max Planck Institute of Experimental Medicine, Max Planck Society;

/persons/resource/persons182104

Brose,  Nils
Molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Citation

Junge, H. J., Rhee, J. S., Jahn, O., Varoqueaux, F., Spiess, J., Waxham, M. N., et al. (2004). Calmodulin and Munc13 Form a Ca2+ Sensor/Effector Complex that Controls Short-Term Synaptic Plasticity. Cell, 118(3), 389-401. doi:10.1016/j.cell.2004.06.029.


Cite as: https://hdl.handle.net/11858/00-001M-0000-0029-30BF-5
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