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Phosphoproteomic profiling of the signaling output of FLT3-ITD and its AC220-resistant mutants reveals profound signaling differences and differential responsiveness to inhibition of downstream kinases.

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Urlaub,  H.
Research Group of Bioanalytical Mass Spectrometry, MPI for biophysical chemistry, Max Planck Society;

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Doebele, C., Kovar, J., Yepes, D., Münch, S., Schnütgen, F., Bohnenberger, H., et al. (2015). Phosphoproteomic profiling of the signaling output of FLT3-ITD and its AC220-resistant mutants reveals profound signaling differences and differential responsiveness to inhibition of downstream kinases. Blood, 126(23): 2453.


Cite as: http://hdl.handle.net/11858/00-001M-0000-0029-D35A-1
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