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Journal Article

Mammalian cadherins and protocadherins: about cell death, synapses and processing

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Junghans,  Dirk
Emeritus Group: Molecular Embryology, Max Planck Institute of Immunobiology and Epigenetics, Max Planck Society;

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Haas,  Ingrid G.
Department of Molecular Embryology, Max Planck Institute of Immunobiology and Epigenetics, Max Planck Society;

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Kemler,  Rolf
Emeritus Group: Molecular Embryology, Max Planck Institute of Immunobiology and Epigenetics, Max Planck Society;

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Citation

Junghans, D., Haas, I. G., & Kemler, R. (2005). Mammalian cadherins and protocadherins: about cell death, synapses and processing. Current Opinion in Cell Biology, 17, 1-7.


Cite as: https://hdl.handle.net/11858/00-001M-0000-002B-9394-0
Abstract
Cadherins have been known for a long time to be key elements in many important biological processes. In particular, the role of classical cadherins in mediating adhesion has been examined in great detail. Over recent years, the accumulation of experimental tools and mice mutants has allowed more refined analysis of cadherin functions, and new aspects such as signaling and synapse dynamics have become the center of interest. In addition, the study of mice lacking the entire protocadherin-γ cluster shed the first light on a possible novel function of members of this cadherin family in synapse formation and cell survival during development.