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Synaptic UNC13A protein variant causes increased neurotransmission and dyskinetic movement disorder

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Lipstein,  Noa
Molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Jahn,  Olaf
Proteomics, Wiss. Servicegruppen, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Taschenberger,  Holger
Molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Rhee,  JeongSeop
Neurophysiology of synapse, Molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Brose,  Nils
Molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Citation

Lipstein, N., Verhoeven-Duif, N. M., Michelassi, F. E., Calloway, N., van Hasselt, P. M., Pienkowska, K., et al. (2017). Synaptic UNC13A protein variant causes increased neurotransmission and dyskinetic movement disorder. The Journal of Clinical Investigation, 127(3), 1005-1018. doi:10.1172/JCI90259.


Cite as: http://hdl.handle.net/11858/00-001M-0000-002D-704D-B
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