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Potentiation of glycine-gated NR1/NR3A NMDA receptors relieves Ca2+-dependent outward rectification

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Madry,  C.
Neurochemistry Department, Max Planck Institute for Brain Research, Max Planck Society;

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Betz,  H.
Neurochemistry Department, Max Planck Institute for Brain Research, Max Planck Society;

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Geiger,  J. R. P.
Neurochemistry Department, Max Planck Institute for Brain Research, Max Planck Society;

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Laube,  B.
Neurochemistry Department, Max Planck Institute for Brain Research, Max Planck Society;

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Madry, C., Betz, H., Geiger, J. R. P., & Laube, B. (2010). Potentiation of glycine-gated NR1/NR3A NMDA receptors relieves Ca2+-dependent outward rectification. Frontiers in Molecular Neuroscience, 3: 6.


Cite as: https://hdl.handle.net/11858/00-001M-0000-002E-217A-F
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