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Aberrant splicing of the tumor suppressor CYLD promotes the development of chronic lymphocytic leukemia via sustained NF-kappa B signaling

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Al-Maarri,  Mona
Obesity and Cancer, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Vogt,  Merly C.
Neuronal Control of Metabolism, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Wunderlich,  Frank Thomas
Obesity and Cancer, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Hahn, M., Buerckert, J.-P., Luttenberger, C., Klebow, S., Hess, M., Al-Maarri, M., Vogt, M. C., Reißig, S., Hallek, M., Wienecke-Baldacchino, A., Buch, T., Muller, C., Pallasch, C., Wunderlich, F. T., Waisman, A., & Hoevelmeyer, N. (2018). Aberrant splicing of the tumor suppressor CYLD promotes the development of chronic lymphocytic leukemia via sustained NF-kappa B signaling. Leukemia: the Journal of Normal and Malignant Hemopoiese; Official Journal of the Leukemia Research Fund U.K., 32(1), 72-82. doi:10.1038/leu.2017.168.


引用: https://hdl.handle.net/21.11116/0000-0000-3F43-F
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