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Glutamatergic nervous system degeneration in a C. elegans TauA152T tauopathy model involves pathways of excitotoxicity and Ca2+ dysregulation

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Mandelkow,  Eckhard
Eckhard Mandelkow, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Mandelkow,  Eva-Maria
Eckhard Mandelkow, Emeriti, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Pir,  Gulam Jeelani
Neuronal Control of Metabolism, Department Jens Brüning, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Citation

Choudhary, B., Mandelkow, E., Mandelkow, E.-M., & Pir, G. J. (2018). Glutamatergic nervous system degeneration in a C. elegans TauA152T tauopathy model involves pathways of excitotoxicity and Ca2+ dysregulation. Neurobiology of Disease, 117, 189-202. doi:10.1016/j.nbd.2018.06.005.


Cite as: http://hdl.handle.net/21.11116/0000-0001-9C05-A
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