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Journal Article

MiR221 promotes precursor B-cell retention in the bone marrow by amplifying the PI3K-signaling pathway in mice

MPS-Authors
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Petkau,  Georg
Max-Planck Fellowship Lymphocyte Development, Max Planck Institute for Infection Biology, Max Planck Society;

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Kawano,  Yohei
Max-Planck Fellowship Lymphocyte Development, Max Planck Institute for Infection Biology, Max Planck Society;

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Wolf,  Inge
Max-Planck Fellowship Lymphocyte Development, Max Planck Institute for Infection Biology, Max Planck Society;

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Melchers,  Fritz
Max-Planck Fellowship Lymphocyte Development, Max Planck Institute for Infection Biology, Max Planck Society;

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Fulltext (public)

Eur_J_Immunol_2018_48_975.pdf
(Publisher version), 3MB

Supplementary Material (public)
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Citation

Petkau, G., Kawano, Y., Wolf, I., Knoll, M., & Melchers, F. (2018). MiR221 promotes precursor B-cell retention in the bone marrow by amplifying the PI3K-signaling pathway in mice. European Journal of Immunology, 48(6), 975-989. doi:10.1002/eji.201747354.


Cite as: http://hdl.handle.net/21.11116/0000-0001-BDBF-4
Abstract
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