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Opposite genetic effects for polygenic ASD risk shared with and independent of ADHD: Evidence for a cancelling-out hypothesis studying genetic overlap with language and literacy

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Verhoef,  Ellen
Language and Genetics Department, MPI for Psycholinguistics, Max Planck Society;
International Max Planck Research School for Language Sciences, MPI for Psycholinguistics, Max Planck Society;
Population genetics of human communication, MPI for Psycholinguistics, Max Planck Society;

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Shapland,  Chin Yang
Language and Genetics Department, MPI for Psycholinguistics, Max Planck Society;

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Fisher,  Simon E.
Language and Genetics Department, MPI for Psycholinguistics, Max Planck Society;
Donders Institute for Brain, Cognition and Behaviour, Radboud University, Nijmegen, The Netherlands;

St Pourcain,  Beate
Language and Genetics Department, MPI for Psycholinguistics, Max Planck Society;
MRC Integrative Epidemiology Unit, University of Bristol, Bristol, UK;
Donders Institute for Brain, Cognition and Behaviour, Radboud University, Nijmegen, The Netherlands;
Population genetics of human communication, MPI for Psycholinguistics, Max Planck Society;

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Citation

Verhoef, E., Grove, J., Shapland, C. Y., Demontis, D., Burgess, S., iPSYCH-Broad-PGC ASD Consortium, et al. (2018). Opposite genetic effects for polygenic ASD risk shared with and independent of ADHD: Evidence for a cancelling-out hypothesis studying genetic overlap with language and literacy. Talk presented at the 26th World Congress of Psychiatric Genetics 2018. Glasgow, UK. 2018-10-11 - 2018-11-15.


Cite as: http://hdl.handle.net/21.11116/0000-0003-43B7-3
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