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PAR-3 controls endothelial planar polarity and vascular inflammation under laminar flow

MPS-Authors
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Hikita,  Takao
Cell Polarity and Organogenesis, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Mirzapourshafiyi,  Fatemeh
Cell Polarity and Organogenesis, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons239434

Riddell,  Meghan
Cell Polarity and Organogenesis, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons239437

Pasha,  Ayesha
Cell Polarity and Organogenesis, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Li,  Mengnan
Cell Polarity and Organogenesis, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons239436

Kawamura,  Takuji
Cell Polarity and Organogenesis, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons224376

Nakayama,  Masanori
Cell Polarity and Organogenesis, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Citation

Hikita, T., Mirzapourshafiyi, F., Barbacena, P., Riddell, M., Pasha, A., Li, M., et al. (2018). PAR-3 controls endothelial planar polarity and vascular inflammation under laminar flow. EMBO REPORTS, 19(9): e45253. doi:10.15252/embr.201745253.


Cite as: http://hdl.handle.net/21.11116/0000-0004-5F0D-5
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