English
 
Help Privacy Policy Disclaimer
  Advanced SearchBrowse

Item

ITEM ACTIONSEXPORT

Released

Journal Article

Testing hypotheses about the underlying deficit of Apraxia of Speech (AOS) through computational neural modelling with the DIVA model

MPS-Authors
/persons/resource/persons192404

Rodd,  Joe
Psychology of Language Department, MPI for Psycholinguistics, Max Planck Society;
Centre for Language Studies, Radboud University;
International Max Planck Research School for Language Sciences, MPI for Psycholinguistics, Max Planck Society;

External Resource
No external resources are shared
Fulltext (restricted access)
There are currently no full texts shared for your IP range.
Supplementary Material (public)
There is no public supplementary material available
Citation

Terband, H., Rodd, J., & Maas, E. (2020). Testing hypotheses about the underlying deficit of Apraxia of Speech (AOS) through computational neural modelling with the DIVA model. International Journal of Speech-Language Pathology, 22(4), 475-486. doi:10.1080/17549507.2019.1669711.


Cite as: https://hdl.handle.net/21.11116/0000-0004-C256-0
Abstract
Purpose: A recent behavioural experiment featuring a noise masking paradigm suggests that Apraxia of Speech (AOS) reflects a disruption of feedforward control, whereas feedback control is spared and plays a more prominent role in achieving and maintaining segmental contrasts. The present study set out to validate the interpretation of AOS as a possible feedforward impairment using computational neural modelling with the DIVA (Directions Into Velocities of Articulators) model.

Method: In a series of computational simulations with the DIVA model featuring a noise-masking paradigm mimicking the behavioural experiment, we investigated the effect of a feedforward, feedback, feedforward + feedback, and an upper motor neuron dysarthria impairment on average vowel spacing and dispersion in the production of six/bVt/speech targets.

Result: The simulation results indicate that the output of the model with the simulated feedforward deficit resembled the group findings for the human speakers with AOS best.

Conclusion: These results provide support to the interpretation of the human observations, corroborating the notion that AOS can be conceptualised as a deficit in feedforward control.