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Journal Article

FX11 limits Mycobacterium tuberculosis growth and potentiates bactericidal activity of isoniazid through host-directed activity

MPS-Authors
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Krishnamoorthy,  Gopinath
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Kaiser,  Peggy
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Abu Abed,  Ulrike
Core Facilities / Microscopy, Max Planck Institute for Infection Biology, Max Planck Society;

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Weiner,  January III
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Alves,  Pedro
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Brinkmann,  Volker
Core Facilities / Microscopy, Max Planck Institute for Infection Biology, Max Planck Society;

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Kaufmann,  Stefan H. E.
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Dis_Model_Mech_2020_13_41954.pdf
(Publisher version), 4MB

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Citation

Krishnamoorthy, G., Kaiser, P., Abu Abed, U., Weiner, J. I., Alves, P., Brinkmann, V., et al. (2020). FX11 limits Mycobacterium tuberculosis growth and potentiates bactericidal activity of isoniazid through host-directed activity. Disease Models & Mechanisms, 13(3): dmm041954. doi: 10.1242/dmm.041954.


Cite as: https://hdl.handle.net/21.11116/0000-0006-872D-0
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