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Fibrillin-2 is a key mediator of smooth muscle extracellular matrix homeostasis during mouse tracheal tubulogenesis

MPS-Authors
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Yin,  Wenguang
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Kim,  Hyun-Taek
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Wang,  ShengPeng
Pharmacology, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Gunawan,  Felix
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Li,  Rui
Pharmacology, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Buettner,  Carmen
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Grohmann,  Beate
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Offermanns,  Stefan
Pharmacology, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Stainier,  Didier Y. R.
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Citation

Yin, W., Kim, H.-T., Wang, S., Gunawan, F., Li, R., Buettner, C., et al. (2019). Fibrillin-2 is a key mediator of smooth muscle extracellular matrix homeostasis during mouse tracheal tubulogenesis. EUROPEAN RESPIRATORY JOURNAL, 53(3): 1800840. doi:10.1183/13993003.00840-2018.


Cite as: http://hdl.handle.net/21.11116/0000-0006-AD47-8
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