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Inactivation of nuclear histone deacetylases by EP300 disrupts the MiCEE complex in idiopathic pulmonary fibrosis

MPS-Authors
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Rubio,  Karla
Lung Cancer Epigenetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Singh,  Indrabahadur
Lung Cancer Epigenetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons224346

Dobersch,  Stephanie
Lung Cancer Epigenetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Sarvari,  Pouya
Lung Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Guenther,  Stefan
Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Cordero,  Julio
Lung Cancer Epigenetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons224348

Mehta,  Aditi
Lung Cancer Epigenetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons224334

Seeger,  Werner
Lung Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Savai,  Rajkumar
Lung Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons224354

Dobreva,  Gergana
Origin of Cardiac Cell Lineages, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Savai-Pullamsetti,  Soni
Lung Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Braun,  Thomas
Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Barreto,  Guillermo
Lung Cancer Epigenetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Citation

Rubio, K., Singh, I., Dobersch, S., Sarvari, P., Guenther, S., Cordero, J., et al. (2019). Inactivation of nuclear histone deacetylases by EP300 disrupts the MiCEE complex in idiopathic pulmonary fibrosis. NATURE COMMUNICATIONS, 10: 2229. doi:10.1038/s41467-019-10066-7.


Cite as: https://hdl.handle.net/21.11116/0000-0006-AF95-D
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