RASSF10 is frequently epigenetically inactivated in kidney cancer and its 
knockout promotes neoplasia in cancer prone mice

Richter, Antje M.; Woods, Michelle L.; Kuester, Miriam M.; Walesch, Sara K.; Braun, Thomas; Boettger, Thomas; Dammann, Reinhard H.

doi:10.1038/s41388-020-1195-6

Item

ITEM ACTIONSEXPORT

Add to Basket

Local TagsRelease HistoryDetailsSummary

Released

Journal Article

RASSF10 is frequently epigenetically inactivated in kidney cancer and its knockout promotes neoplasia in cancer prone mice

MPS-Authors

/persons/resource/persons248833

Richter, Antje M.
Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons224052

Braun, Thomas
Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons224044

Boettger, Thomas
Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

External Resource

No external resources are shared

Fulltext (restricted access)

There are currently no full texts shared for your IP range.

Fulltext (public)

There are no public fulltexts stored in PuRe

Supplementary Material (public)

There is no public supplementary material available

Citation

Richter, A. M., Woods, M. L., Kuester, M. M., Walesch, S. K., Braun, T., Boettger, T., et al. (2020). RASSF10 is frequently epigenetically inactivated in kidney cancer and its knockout promotes neoplasia in cancer prone mice. ONCOGENE, 39(15), 3114-3127. doi:10.1038/s41388-020-1195-6.

Cite as: https://hdl.handle.net/21.11116/0000-0006-B226-6

Abstract

There is no abstract available