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AP-1 Contributes to Chromatin Accessibility to Promote Sarcomere Disassembly and Cardiomyocyte Protrusion During Zebrafish Heart Regeneration

MPS-Authors
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Beisaw,  Arica
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Kuenne,  Carsten
Bioinformatics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons224128

Guenther,  Stefan
Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Dallmann,  Julia
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Wu,  Chi-Chung
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Bentsen,  Mette
Bioinformatics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Looso,  Mario
Bioinformatics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

/persons/resource/persons224278

Stainier,  Didier Y. R.
Developmental Genetics, Max Planck Institute for Heart and Lung Research, Max Planck Society;

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Citation

Beisaw, A., Kuenne, C., Guenther, S., Dallmann, J., Wu, C.-C., Bentsen, M., et al. (2020). AP-1 Contributes to Chromatin Accessibility to Promote Sarcomere Disassembly and Cardiomyocyte Protrusion During Zebrafish Heart Regeneration. CIRCULATION RESEARCH, 126(12), 1760-1778. doi:10.1161/CIRCRESAHA.119.316167.


Cite as: http://hdl.handle.net/21.11116/0000-0006-C507-4
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