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Soluble beta-amyloid1-40 induces NMDA-dependent degradation of postsynaptic density-95 at glutamatergic synapses.

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Tirard,  Marilyn
Molecular neurobiology, Max Planck Institute of Experimental Medicine, Max Planck Society;

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Roselli, F., Tirard, M., Lu, J., Hutzler, P., Lamberti, P., Livrea, P., et al. (2005). Soluble beta-amyloid1-40 induces NMDA-dependent degradation of postsynaptic density-95 at glutamatergic synapses. The Journal of Neuroscience, 25(48), 11061-11070.


Cite as: https://hdl.handle.net/21.11116/0000-0007-1608-8
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