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Author correction: Voltage-independent GluN2A-type NMDA receptor Ca2+ signaling promotes audiogenic seizures, attentional and cognitive deficits in mice

MPS-Authors
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Bertocchi,  Ilaria
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Rozov,  Andrej
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Bus,  Thorsten
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Pawlak,  Verena
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;
Department of Behavior and Brain Organization, Center of Advanced European Studies and Research (caesar), Max Planck Society;

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Serafino,  Marta
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Sonntag,  Hannah
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Yang,  Boyi
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Burnashev,  Nail
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Li,  Shi-Bin
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Obenhaus,  Horst A.
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Single,  Frank N.
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Köhr,  Georg
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

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Sprengel,  Rolf
Department of Molecular Neurobiology, Max Planck Institute for Medical Research, Max Planck Society;

External Resource

https://www.nature.com/articles/s42003-021-01717-x
(Publisher version)

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s42003-021-01717-x.pdf
(Publisher version), 533KB

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Citation

Bertocchi, I., Eltokhi, A., Rozov, A., Chi, V. N., Jensen, V., Bus, T., et al. (2021). Author correction: Voltage-independent GluN2A-type NMDA receptor Ca2+ signaling promotes audiogenic seizures, attentional and cognitive deficits in mice. Communications Biology, 4: 187. doi:10.1038/s42003-020-01538-4.


Cite as: https://hdl.handle.net/21.11116/0000-0007-F441-C
Abstract
The original version of this Article contained an error in the variant mentioned in the following text from the “Introduction”: “Notably, a similar Mg2+ block attenuating point mutation (c.1841A>G, p.Asn615Ser) at the identical position of the GluN2A subunit was found in two unrelated young female patients who suffered from epileptic seizures...”. The correct variant is (c.1845C>A, p.Asn615Lys).

The original version of this Article also contained an error in the mutation mentioned in the following text from the “Results” section “Generation of GluNA (N615S)-expressing mice”: “By classical gene-targeted replacement28 we inserted the c.1841A>G mutation at the homologous position in exon 10…”. The correct position of this mutation is c.1844A>G.