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Journal Article

Cellular stress promotes NOD1/2-dependent inflammation via the endogenous metabolite sphingosine-1-phosphate

MPS-Authors
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Pei,  Gang
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Zyla,  Joanna
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Alves,  Pedro
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Saikali,  Philippe
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Lozza,  Laura
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Nieuwenhuizen,  Natalie
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Weiner,  January
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Mollenkopf,  Hans-Joachim
Core Facilities / Microarray, Max Planck Institute for Infection Biology, Max Planck Society;

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Kaufmann,  Stefan H. E.
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Fulltext (public)

embj.2020106272.pdf
(Publisher version), 2MB

Supplementary Material (public)
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Citation

Pei, G., Zyla, J., He, L., Alves, P., Steinle, H., Saikali, P., et al. (2021). Cellular stress promotes NOD1/2-dependent inflammation via the endogenous metabolite sphingosine-1-phosphate. EMBO Journal, e106272. doi:10.15252/embj.2020106272.


Cite as: http://hdl.handle.net/21.11116/0000-0008-8481-0
Abstract
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