MiR-132 controls pancreatic beta cell proliferation and survival through 
Pten/Akt/Foxo3 signaling.

Mziaut, Hassan; Henniger, Georg; Ganss, Katharina; Hempel, Sebastian; Wolk, S; McChord, Johanna; Chowdhury, Kamal; Ravassard, Philippe; Knoch, Klaus-Peter; Krautz, Christian; Weitz, Jürgen; Grützmann, Robert; Pilarsky, Christian; Solimena, Michele; Kersting, Stephan

doi:10.1016/j.molmet.2019.11.012

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MiR-132 controls pancreatic beta cell proliferation and survival through Pten/Akt/Foxo3 signaling.

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Solimena, Michele
Max Planck Institute for Molecular Cell Biology and Genetics, Max Planck Society;

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Citation

Mziaut, H., Henniger, G., Ganss, K., Hempel, S., Wolk, S., McChord, J., et al. (2020). MiR-132 controls pancreatic beta cell proliferation and survival through Pten/Akt/Foxo3 signaling. Molecular metabolism, 31, 150-162. doi:10.1016/j.molmet.2019.11.012.

Cite as: https://hdl.handle.net/21.11116/0000-0008-A2F8-9

Abstract

MicroRNAs (miRNAs) play an integral role in maintaining beta cell function and identity. Deciphering their targets and precise role, however, remains challenging. In this study, we aimed to identify miRNAs and their downstream targets involved in the regeneration of islet beta cells following partial pancreatectomy in mice.