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Deletion of Tbc1d4/As160 abrogates cardiac glucose uptake and increases myocardial damage after ischemia/reperfusion

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Weiss,  Jonathan
Funktionelle Krebsgenomforschung, Junior Research Groups, Max-Planck-Institut für neurologische Forschung, Managing Director: D. Yves von Cramon, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Cremer,  Anna Lena
Multimodal Imaging of Brain Metabolism, Research Groups, Max Planck Institute for Metabolism Research, Managing Director: Jens Brüning, Max Planck Society;

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Backes,  Heiko
Backes, Wiedermann – Multimodal Imaging, Scientific Services and Development, Max Planck Institute for Metabolism Research, Max Planck Society;

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Binsch, C., Barbosa, D., Hansen-Dille, G., Hubert, M., Hodge, S., Kolasa, M., et al. (2023). Deletion of Tbc1d4/As160 abrogates cardiac glucose uptake and increases myocardial damage after ischemia/reperfusion. Cardiovascular Diabetology, 22(1): 17. doi:10.1186/s12933-023-01746-2.


Cite as: https://hdl.handle.net/21.11116/0000-000C-E754-1
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